Candida albicans commensalism in the oral mucosa is favoured by limited virulence and metabolic adaptation | PLOS Pathogens
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C. albicans yeasts were added to TR146 monolayers and incubated under... | Download Scientific Diagram
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The Candida albicans toxin candidalysin mediates distinct epithelial inflammatory responses through p38 and EGFR-ERK pathways | Science Signaling
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Activation of NF-κB and MAPK signaling by different Candida species and... | Download Scientific Diagram
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Cellular interactions of Candida albicans with human oral epithelial cells and enterocytes - Dalle - 2010 - Cellular Microbiology - Wiley Online Library
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Pathogens | Free Full-Text | Candida albicans Interactions with Mucosal Surfaces during Health and Disease
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Frontiers | Candida albicans Sap6 Initiates Oral Mucosal Inflammation via the Protease Activated Receptor PAR2
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Cellular interactions of Candida albicans with human oral epithelial cells and enterocytes - Dalle - 2010 - Cellular Microbiology - Wiley Online Library
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Frontiers | Candida albicans Sap6 Initiates Oral Mucosal Inflammation via the Protease Activated Receptor PAR2
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Candidalysin activates innate epithelial immune responses via epidermal growth factor receptor | Nature Communications
Processing of <italic toggle='yes'>Candida albicans</italic> Ece1p Is Critical for Candidalysin Maturation a
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ERG3 and ERG11 genes are critical for the pathogenesis of Candida albicans during the oral mucosal infection | International Journal of Oral Science
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Candida albicans Cell Wall Glycosylation May Be Indirectly Required for Activation of Epithelial Cell Proinflammatory Responses | Infection and Immunity
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